A group of Italian researchers have found that mice lacking a protein called AT1A live substantially longer than normal mice.
Ariela Benigni, of the "Mario Negri" Institute for Pharmacological Research, points out that medicines that antagonize AT1A are currently used to treat high blood pressure.
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Reducing the levels of a protein called tau helped in the reduction of Alzheimer’s symptoms in mice.
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According to Benigni, the research team's analysis also suggests that the increased lifespan in mice lacking AT1A was likely a result of decreased oxidative damage to cells, a key factor in ageing, and increased levels of genes involved in cell survival.
Source-ANI
ARU/L
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