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The research team studied three mouse models of kidney fibrosis.
One group of mice contained HIV viral proteins incorporated into their genome.
The second group was injected with a high dose of folic acid.
In the third group, kidney filtration was blocked in one kidney.
All of these factors caused kidney fibrosis, Xinhua reported.
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They found that HIPK2, a protein kinase, or regulator, was highly active in the mice with kidney fibrosis.
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The study was published this week in Nature Medicine.
Source-IANS