A brain mechanism that switches off traumatic feelings associated with bad memories has been identified by researchers from University of California - Irvine and the University of Muenster in Germany. The finding may lead to new drugs for panic disorders
They found that a small brain protein called neuropeptide S is involved in erasing traumatic responses to adverse memories by working on a tiny group of neurons inside the amygdala where those memories are stored.
Trauma Care
Accident and trauma care is an important public health challenge that requires immediate attention. Injury (trauma) is the leading cause of death for all age groups under the age of 44.
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"The exciting part of this study is that we have discovered a completely new process that regulates the adverse responses to bad memories," said Rainer Reinscheid, pharmacology and pharmaceutical sciences associate professor at UCI.
"These findings can help the development of new drugs to treat conditions in which people are haunted by persistent fears, such as posttraumatic stress disorder or other panic disorders," Reinscheid added.
![Head Injury]( "Head Injury")
Head injury or traumatic brain injury is a leading cause of disability among children and young adults that leads to varying degrees of physical and mental stability. Fortunately, it is also one of the preventable brain disorder.
During the study, researchers exposed mice to situations that caused adverse memories.
The researchers saw that when NPS receptors in amygdala neurons are blocked, the traumatic responses to bad memories persisted longer.
In turn, when researchers treated the mice with compounds activating these receptors, traumatic responses disappeared faster.
The study appears in the July 31 issue of Neuron.
Source: ANI
RAS/L
Head Injury
Head injury or traumatic brain injury is a leading cause of disability among children and young adults that leads to varying degrees of physical and mental stability. Fortunately, it is also one of the preventable brain disorder.
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During the study, researchers exposed mice to situations that caused adverse memories.
The researchers saw that when NPS receptors in amygdala neurons are blocked, the traumatic responses to bad memories persisted longer.
In turn, when researchers treated the mice with compounds activating these receptors, traumatic responses disappeared faster.
The study appears in the July 31 issue of Neuron.
Source: ANI
RAS/L
Majority of Psychiatric Patients Suffer from More Than One Disorders
Researchers at the Rhode Island Hospital have found that most psychiatric patients have more than one diagnosis.
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 Why Traumatic Events are Not Erased from Mind?Emotional memories of a traumatic episode in life often persist in our minds. | Advertisement
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