Medindia » Consumer Health » Health Information » Latest Publication and Research on Alcoholic Liver Disease

Alcoholic Liver Disease

Latest Publication and Research on Alcoholic Liver Disease

RSS Feeds   Text 
  2011 Dec 3  

Cytokeratin-18 and hyaluronic acid levels predict liver fibrosis in children with non-alcoholic fatty liver disease.

Lebensztejn DM, Wierzbicka A, Socha P, Pronicki M, Skiba E, Werpachowska I, Kaczmarski M

Department of Pediatrics, Gastroenterology and Allergology, Medical University of Bialystok, Bialystok, Poland.

Objectives: There is a need to replace liver biopsy with non-invasive markers that predict the degree of liver fibrosis in fatty liver disease related... Read More

Source: PubMed

  2011 Dec 5  

Cumulative risk of cardiovascular events following orthotopic liver transplantation.

Albeldawi M, Ashish Aggarwal, Madhwal S, Cywinski J, Lopez R, Eghtesad B, Zein NN

Department of Gastroenterology and Hepatology, Cleveland, Ohio, USA.

As post-orthotopic liver transplantation (OLT) survival improves, cardiovascular (CV) disease has emerged as the leading cause of non-graft related de... Read More

Source: PubMed

Ther Umsch    

[Liver transplantation - when and for whom it should be performed].

Morell B, Dufour JF

Universitätsklinik für Viszerale Chirurgie und Medizin, Inselspital Bern.

During the past two decades, orthotopic liver transplantation (OLT) emerged to the treatment of choice for patients with end-stage liver disease. In S... Read More

Source: PubMed

Nihon Shokakibyo Gakkai Zasshi    

A case of non-alcoholic fatty liver disease (NAFLD) aggravated after treatment with raloxifene.

Matsumura M, Tashiro K, Miura A, Tajima T, Kinoshita I, Kojima E, Yoshizawa A

Department of Gastroenterology, Nagano Chuo Hospital.

A 70-year-old woman presented with abnormal liver function test 3 months after the introduction of daily intake of raloxifene, a selective estrogen re... Read More

Source: PubMed

  2011 Dec 4  

Specificity of unenhanced CT for non-invasive diagnosis of hepatic steatosis: implications for the investigation of the natural history of incidental steatosis.

Pickhardt PJ, Park SH, Hahn L, Lee SG, Bae KT, Yu ES

Department of Radiology, University of Wisconsin School of Medicine & Public Health, E3/311 Clinical Science Center, 600 Highland Ave., Madison, WI, 53792-3252, USA.

OBJECTIVES: To determine a highly specific liver attenuation threshold at unenhanced CT for biopsy-proven moderate to severe hep... Read More

Source: PubMed

  2011 Dec 4  

Overexpression of 11ß-hydroxysteroid dehydrogenase type 1 in visceral adipose tissue and portal hypercortisolism in non-alcoholic fatty liver disease.

Candia R, Riquelme A, Baudrand R, Carvajal CA, Morales M, Solís N, Pizarro M, Escalona A, Carrasco G, Boza C, Pérez G, Padilla O, Cerda J, Fardella CE, Arrese M

Department of Gastroenterology, Pontificia Universidad Católica de Chile, Santiago, Chile.

BACKGROUND: The enzyme 11ß-hydroxysteroid-dehydrogenase type 1 (11ß-HSD1) catalyses the reactivation of intracellular cortisol. We explored the potent... Read More

Source: PubMed

  2011 Dec 1  

Bile duct changes in different etiologic types of end-stage chronic liver disease: a study on native explant livers.

Jain D, Nayak NC

Department of Pathology, Sir Ganga Ram Hospital, New Delhi, India.

AimsBile duct changes in the form of intraepithelial neoplasia or dysplasia have been well studied in chronic biliary tract diseases. It is important ... Read More

Source: PubMed

  2011 Nov 29  

Influence of non-alcoholic fatty liver disease on the prognosis in patients with colorectal cancer.

Min YW, Yun HS, Chang WI, Kim JY, Kim YH, Son HJ, Kim JJ, Rhee JC, Chang DK

Division of Gastroenterology and Hepatology, Department of Medicine, Samsung Medical Center, Sungkyunkwan University School of Medicine, 50, Irwon-dong, Gangnam-gu, Seoul, 135-710, Republic of Korea.

BACKGROUND/AIM: Non-alcoholic fatty liver disease (NAFLD) is associated with multiple adenomatous polyps and advanced neoplasm. This study aims to inv... Read More

Source: PubMed

PLoS ONE    

Synergy Analysis Reveals Association between Insulin Signaling and Desmoplakin Expression in Palmitate Treated HepG2 Cells.

Wang X, Nath A, Yang X, Portis A, Walton SP, Chan C

Department of Chemical Engineering and Materials Science, Michigan State University, East Lansing, Michigan, United States of America.

The regulation of complex cellular activities in palmitate treated HepG2 cells, and the ensuing cytotoxic phenotype, involves cooperative interactions... Read More

Source: PubMed

  2011 Dec 3  

Cytokeratin-18 and hyaluronic acid levels predict liver fibrosis in children with non-alcoholic fatty liver disease.

Lebensztejn DM, Wierzbicka A, Socha P, Pronicki M, Skiba E, Werpachowska I, Kaczmarski M

Department of Pediatrics, Gastroenterology and Allergology, Medical University of Bialystok, Bialystok, Poland.

Objectives: There is a need to replace liver biopsy with non-invasive markers that predict the degree of liver fibrosis in fatty liver disease related... Read More

Source: PubMed

  2011 Dec 5  

Cumulative risk of cardiovascular events following orthotopic liver transplantation.

Albeldawi M, Ashish Aggarwal, Madhwal S, Cywinski J, Lopez R, Eghtesad B, Zein NN

Department of Gastroenterology and Hepatology, Cleveland, Ohio, USA.

As post-orthotopic liver transplantation (OLT) survival improves, cardiovascular (CV) disease has emerged as the leading cause of non-graft related de... Read More

Source: PubMed

Ther Umsch    

[Liver transplantation - when and for whom it should be performed].

Morell B, Dufour JF

Universitätsklinik für Viszerale Chirurgie und Medizin, Inselspital Bern.

During the past two decades, orthotopic liver transplantation (OLT) emerged to the treatment of choice for patients with end-stage liver disease. In S... Read More

Source: PubMed

Nihon Shokakibyo Gakkai Zasshi    

A case of non-alcoholic fatty liver disease (NAFLD) aggravated after treatment with raloxifene.

Matsumura M, Tashiro K, Miura A, Tajima T, Kinoshita I, Kojima E, Yoshizawa A

Department of Gastroenterology, Nagano Chuo Hospital.

A 70-year-old woman presented with abnormal liver function test 3 months after the introduction of daily intake of raloxifene, a selective estrogen re... Read More

Source: PubMed

  2011 Dec 4  

Specificity of unenhanced CT for non-invasive diagnosis of hepatic steatosis: implications for the investigation of the natural history of incidental steatosis.

Pickhardt PJ, Park SH, Hahn L, Lee SG, Bae KT, Yu ES

Department of Radiology, University of Wisconsin School of Medicine & Public Health, E3/311 Clinical Science Center, 600 Highland Ave., Madison, WI, 53792-3252, USA.

OBJECTIVES: To determine a highly specific liver attenuation threshold at unenhanced CT for biopsy-proven moderate to severe hep... Read More

Source: PubMed

  2011 Dec 4  

Overexpression of 11ß-hydroxysteroid dehydrogenase type 1 in visceral adipose tissue and portal hypercortisolism in non-alcoholic fatty liver disease.

Candia R, Riquelme A, Baudrand R, Carvajal CA, Morales M, Solís N, Pizarro M, Escalona A, Carrasco G, Boza C, Pérez G, Padilla O, Cerda J, Fardella CE, Arrese M

Department of Gastroenterology, Pontificia Universidad Católica de Chile, Santiago, Chile.

BACKGROUND: The enzyme 11ß-hydroxysteroid-dehydrogenase type 1 (11ß-HSD1) catalyses the reactivation of intracellular cortisol. We explored the potent... Read More

Source: PubMed

  2011 Dec 1  

Bile duct changes in different etiologic types of end-stage chronic liver disease: a study on native explant livers.

Jain D, Nayak NC

Department of Pathology, Sir Ganga Ram Hospital, New Delhi, India.

AimsBile duct changes in the form of intraepithelial neoplasia or dysplasia have been well studied in chronic biliary tract diseases. It is important ... Read More

Source: PubMed

  2011 Nov 29  

Influence of non-alcoholic fatty liver disease on the prognosis in patients with colorectal cancer.

Min YW, Yun HS, Chang WI, Kim JY, Kim YH, Son HJ, Kim JJ, Rhee JC, Chang DK

Division of Gastroenterology and Hepatology, Department of Medicine, Samsung Medical Center, Sungkyunkwan University School of Medicine, 50, Irwon-dong, Gangnam-gu, Seoul, 135-710, Republic of Korea.

BACKGROUND/AIM: Non-alcoholic fatty liver disease (NAFLD) is associated with multiple adenomatous polyps and advanced neoplasm. This study aims to inv... Read More

Source: PubMed

PLoS ONE    

Synergy Analysis Reveals Association between Insulin Signaling and Desmoplakin Expression in Palmitate Treated HepG2 Cells.

Wang X, Nath A, Yang X, Portis A, Walton SP, Chan C

Department of Chemical Engineering and Materials Science, Michigan State University, East Lansing, Michigan, United States of America.

The regulation of complex cellular activities in palmitate treated HepG2 cells, and the ensuing cytotoxic phenotype, involves cooperative interactions... Read More

Source: PubMed

PLoS ONE    

Coordinated defects in hepatic long chain Fatty Acid metabolism and triglyceride accumulation contribute to insulin resistance in non-human primates.

Kamath S, Chavez AO, Gastaldelli A, Casiraghi F, Halff GA, Abrahamian GA, Davalli AM, Bastarrachea RA, Comuzzie AG, Guardado-Mendoza R, Jimenez-Ceja LM, Mattern V, Paez AM, Ricotti A, Tejero ME, Higgins PB, Rodriguez-Sanchez IP, Tripathy D, Defronzo RA, Dick EJ, Cline GW, Folli F

Department of Medicine/Division of Diabetes. The University of Texas Health Science Center at San Antonio, San Antonio, Texas, United States of America.

Non-Alcoholic fatty liver disease (NAFLD) is characterized by accumulation of triglycerides (TG) in hepatocytes, which may also trigger cirrhosis. The... Read More

Source: PubMed

PLoS ONE    

Coordinated defects in hepatic long chain Fatty Acid metabolism and triglyceride accumulation contribute to insulin resistance in non-human primates.

Kamath S, Chavez AO, Gastaldelli A, Casiraghi F, Halff GA, Abrahamian GA, Davalli AM, Bastarrachea RA, Comuzzie AG, Guardado-Mendoza R, Jimenez-Ceja LM, Mattern V, Paez AM, Ricotti A, Tejero ME, Higgins PB, Rodriguez-Sanchez IP, Tripathy D, Defronzo RA, Dick EJ, Cline GW, Folli F

Department of Medicine/Division of Diabetes. The University of Texas Health Science Center at San Antonio, San Antonio, Texas, United States of America.

Non-Alcoholic fatty liver disease (NAFLD) is characterized by accumulation of triglycerides (TG) in hepatocytes, which may also trigger cirrhosis. The... Read More

Source: PubMed


Share it!
Comment & Contribute
Comments should be on the topic and should not be abusive. Comments are normally moderated and are reviewed after they are posted.
* Your comment can be maximum of 2500 characters

Notify me when reply is posted
I agree to the terms and conditions
  
If you have a question about health related issues, you can now post it in our Ask An Expert section on our community website Medwonders.com and get answers from our panel of experts.
Posted by:  chideria(Guest)  Posted on: 02/03/2012
hi, please do not mind my english.i had the liver alcohol for 2years.i drink alot of alcohol before untill,i had a fever and liver pains the doctor said i should chack my liver which is called l.f.t .so after the test the doctor discover all is not will.is abnormal for all{ALT} {AST} {GGT }{ALP} {ALB}it means my liver is not working will.i had a treatment in china for 2years nothing ,even the doctor is tired of me so i stop going there ,i think i will die ,because i send 4320usd for 2year i don't have money to spend any more.i start pray to god to save me.i stop taking alcohol for long,decide to stop eating oil food,fats,egg,bread,fride food and i stop eating fresh food such like fish,meats e.t.c i worriy so much even my stomach is pain me but i hope one day god will hear me .i start cooking my own food without oil,i useing dry things like dry fish ,fresh vegetables,onions,carrots,with maggi, creyfish,white rice,everyday one type of food and i drinking alot of water each 30mins,,i eat alot of fruits like apples,mango,popo,grape e.t.c i don't takeing oranges because i had a stomach pains.i drinking[ vitamin B complx ]and folic acid ]i giving my self alot of rest to help my liver to be strong.i stop to eat too much ,i eat alittle even if i hungry i eat alittle for 3months,believe me i want to test yesterday after my test the result is normal,i don't believe it, i want to another test is normal .my stomach is not paining me again i feeling good now for one week .the liver can healing by it self,i said to myself in is life again i will never drink alcohol even to make a friend that takes it never,care your health my friend i can help you to tell thanks


Posted by:  rayski(Guest)  Posted on: 02/18/2012
Hello I read your comments on your health problem. I am going through the same thing with a friend here in California, I can't comment on your stage of health but my friends only got 2 or 3 weeks left. The Dr says the liver does not heal it self. His stomach has to be drained every 2 day's He is now turning very yellow. I wish you the best and may God help you back to total health.




Posted by:  odira(Guest)  Posted on: 02/03/2012
hi, please do not mind my english.i had the liver alcohol for 2years.i drink alot of alcohol before untill,i had a fever and liver pains the doctor said i should chack my liver which is called l.f.t .so after the test the doctor discover all is not will.is abnormal for all{ALT} {AST} {GGT }{ALP} {ALB}it means my liver is not working will.i had a treatment in china for 2years nothing ,even the doctor is tired of me so i stop going there ,i think i will die ,because i send 4320usd for 2year i don't have money to spend any more.i start pray to god to save me.i stop taking alcohol for long,decide to stop eating oil food,fats,egg,bread,fride food and i stop eating fresh food such like fish,meats e.t.c i worriy so much even my stomach is pain me but i hope one day god will hear me .i start cooking my own food without oil,i useing dry things like dry fish ,fresh vegetables,onions,carrots,with maggi, creyfish,white rice,everyday one type of food and i drinking alot of water each 30mins,,i eat alot of fruits like apples,mango,popo,grape e.t.c i don't takeing oranges because i had a stomach pains.i drinking[ vitamin B complx ]and folic acid ]i giving my self alot of rest to help my liver to be strong.i stop to eat too much ,i eat alittle even if i hungry i eat alittle for 3months,believe me i want to test yesterday after my test the result is normal,i don't believe it, i want to another test is normal .my stomach is not paining me again i feeling good now for one week .the liver can healing by it self,i said to myself in is life again i will never drink alcohol even to make a friend that takes it never.add me for yahoo for more informations about it add me care your health my friend i can help you to tell thanks



Posted by:  Bhel27(Guest)  Posted on: 09/07/2011
My husband is a heavy drinker of whisky. He consume around 500ml of whisky a night and has been alcoholic for more than 10 yrs and is now suffering from liver disease. He starting noticing a pain in his liver area 4 weeks ago and now showing a jaundice or yellowing of his skin and eyes and so as his stool and urine. Are all these symptoms of a severe stage? I think he's got a hepatitis liver disease which worries me and my sons health. Is this contagious or transferrable if it is how? He completely stop drinking as soon as he starts to see the sign and symptoms and trying to eat more healthier food. For how long would he suffer this disease? And is it still possible to cure this?



Posted by:  Jogrif  Posted on: 06/21/2010
My husband drinks an average of 12 cans of beer a day and has been doing it for 20 years now. His face is now is actually purple with many veins showing and he is yellow around the eyes and has developed a "beer gut". He vomits frequently in the morning and doesn't seem to have much energy at all. Also, his mind is not as sharp as before. He knows and admits he is an alcoholic, but will never quit drinking.

Are these symptoms indicative of Hepatitis Liver or still worse Cirrhosis? Do some people have more tolerance to alcohol and not develop cirrhosis?



Posted by:  Liv52  Posted on: 06/15/2010
Thanks for this informative post. It is a good time to start thinking about the importance of liver treatment with alternative medicines.
After lots of clinical research and tests, it has been proved that Liv.52 leads to an improvement in the architecture of the liver. Hepatic function tests and needle biopsy specimens show a definite improvement in hepatocellular function and structure. Regular intake of Liv.52 guarantees an increase in the number of the total mass of functioning hepatocytes. Also, the liver enzymes return to normal levels following an increase in total proteins, albumin and hemoglobin.

We are direct resellers of Himalaya Liv 52 in Europe.Our website will provide a wealth of information, advice and recommendations on how you can improve your liver function using alternative medicines.



Posted by:  Alex.Gallagher(Guest)  Posted on: 12/01/2011
Should I take it [Liv 52 - known as Livercare in USA] before or after consuming alcohol? My experience has been - it suits me better if I take it a few hours after consuming alcohol. Is it okay then for me to keep it that way?




Posted by:  KUKREJA  Posted on: 10/22/2009
Along with the CNS, the LIVER suffers the most from alcohol consumption. Liver can handle only a certain amount of alcohol in any given time. When we drink quickly, our liver cells have to work overtime to process the alcohol. When this is more than the liver can deal with, the excess is transported to the rest of our organs.

Liver needs water to do its job. As alcohol acts as diuretic (makes us pass urine), it dehydrates and forces the liver to divert water from elsewhere.

When liver is processing alcohol, it produces a substance called ACETALDEHYDE which has a toxic effect on the liver itself, as well as the brain and stomach lining.

Not feeling any side effects from drinking does not mean we are not risking liver damage. Chances are that we will not feel anything happening until this most resilient organ of our body (LIVER) has had enough. And, when that happens, what we have are the conditions, not at all easy to be reversed, like fatty liver, alcoholic hepatitis and cirrhosis.



Posted by:  stevenwinton(Guest)  Posted on: 02/23/2009
I think the very important part of treatment is to stop using alcohol completely. If cirrhosis develops then you will need to manage the complications of cirrhosis. You made need a liver transplant. Some vitamins especially B.complex and folic acid can help reverse malnutrition. Abstinence is the mainstay of treatment; it can prevent further damage from alcoholic liver disease and thus prolong life. Because compliance is problematic, a compassionate team approach is essential.

Steven

Alcoholism Information





Posted by:  Arnold(Guest)  Posted on: 10/25/2008
This is a very nice article. I think alcohol liver disease is rare among youngsters.



Posted by:  christinarosy  Posted on: 06/09/2008
Alcohol is metabolized more slowly than it is absorbed. ************************** christinarosy Alcohol abuse affects millions. This site has a lot of useful information. Alcohol Abuse



Posted by:  guest  Posted on: 05/31/2008
my husband had an artery in his face that burst... the doctors said he has liver hepatitis, is that form of hepatitis contagious, should i be worried about myself and my child? Also, he is consuming alcohol in large amounts and isnt quitting, how long does a patient have before cirrhousis develops ? How long can someone live with this disease


Posted by:  guest  Posted on: 09/05/2008
depending on the length of time,and whatlevel of alcohol your husband consumes the approx gestation period for any diagnostic developmental signsare 10yrs.You didn't say how long he has been drinking,or what his drink is.he obvioustly needs help NOW before further medical/psychosocial issues are present.Idon't know what resources they have in America to tackle this problem but he needs to go into a rehab centre for quite a while.Does he accept that he has a problem or--is he in denial? has it started to effect his mental state,and are you being swept along with his interchanging behaviour patterns.Regards to your question about hep c you can only contaminate yourself/daughter if either one of you make direct contact with his blood.ie:cuts etc.I would be more concerned about the effect on the family dynamics that this disease can create. Get him help NOW! GOODLUCK.




View all Comments (11) 
X
Related Links
Bubbles and Brews - Alcohol Facts
Liver Cancer - Animation
Liver Cancer Treatment - Animation
Yoga Asanas - Halasana
Yoga Asanas - Yogmudra
Yoga Asanas - Naukasan
Alcohol Self Screening Test
Blood Alcohol Level
Liver Biopsy
Gallstones
Splenectomy - Animation
Citing this Health Article
The information on Medindia is factual, unbiased and authentic. If you find the content useful - PLEASE INCLUDE A LINK TO THIS PAGE.

Highlight the text, right-click, and select “copy”. Paste the link into your website, email, or any other HTML document.

News on Alcoholic Liver Disease
Are you a member of Facebook? Become a fan of Medindia and get breaking news delivered to your Facebook page.