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The Sufferers of Alzheimers Disease Specially Display the Memory Loss
The researchers say that their suggestion is based on a study that builds on previous animal research, wherein they could completely prevent Alzheimers disease in mice genetically engineered with a human Alzheimers gene, Mouzheimers, by blocking a single site of cleavage of a molecule called APP for amyloid precursor protein.
Normally, this site on APP is attacked by molecular scissors called caspases, but blocking that process prevented the disease.
In their latest study, the researchers studied human brain tissue, and found that patients suffering from AD showed more of the cleavage process than people of the same age who did not have the disease.
However, upon extending their study to much younger people without Alzheimers disease, they were astonished to find that younger subjects displayed as much as ten times the amount of the same cleavage event as the AD patients.
The researchers say that their study implicates a biochemical switch associated with that cleavage of APP, causing AD brains to become stuck in the process of breaking memories.
The study also points to AD as a syndrome affecting the plasticity or malleability of the brain, they add.
Published in the Journal of Alzheimers Disease, the new study provides new insight into a molecular event resulting in decreased brain plasticity, a central feature of AD.
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