Armon says that focusing on processes at initiation of sporadic ALS, and close to it, may provide new avenues to treatment to stop its progression.
"This has been realized in some animal models of familial ALS, but not in humans. The hope that these concepts may apply to sporadic disease and change its outlook in the future is supported by establishing the association of smoking with ALS occurrence," he added.
The study appears in Neurology, the medical journal of the American Academy of Neurology.
Source-ANI
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