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Smoking Destroys Genes That Prevents Premature Aging
January 24, 2008 at 5:52 PM
Research News
If this gene is absent, one not only loses youthfulness, but the lungs become vulnerable to destructive inflammation and diseases such as chronic obstructive pulmonary disease (COPD) and lung cancer.
The study was led by Irfan Rahman, Ph.D., associate professor of Environmental Medicine and an investigator in the University of Rochester’s Lung Biology and Disease Program.
After identifying the Sirtuin (SIRT1) gene’s role in pulmonary disease, the researchers expected to find ways to restore it and kick-start lung healing. They’ve begun testing the powerful antioxidant resveratrol, extracted from red grape skins, to develop a treatment targeting SIRT1 and reverse lung damage, or at least enhance the way standard COPD therapies work.
“This novel protein will allow us to program our body’s immune-inflammatory system against lung damage and premature aging. The hallmark of this discovery is that we may be able to provide remedies to millions of smokers who would like to quit but cannot kick their addiction, and millions of former smokers who, despite quitting, remain at risk for illness as they age,” said Rahman.
For years, Rahman studied how the 4,700 toxic chemical compounds in cigarettes attack lung tissue. He also focused on why some people seem genetically predisposed to develop lung diseases while others are more fortunate, even after being smokers.
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