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Protein That Grounds Mad-Cow Disease May Also Aid In Sense Of Smell
Dr. Stuart Firestein, who led the study on mice, has revealed that the animals that were bred to lack the prion protein could not find buried food or choose between smells.
He believes that the loss of the protein's original role might be responsible for some of the symptoms of prion disease.
In their study report, the researchers said that the mice without prion-protein, though retained the ability to detect scents, had lost some higher functions which required that smell information to be analysed and processed by the brain.
They revealed that they had observed changes in the communication between neurons in the nerve cells of the olfactory bulb, part of the forebrain that deals with odours.
According to Nature Neuroscience, the researchers found the mice to develop the ability to discriminate between odours when they restored the protein to that part of the brain.
The team agreed that their finding had no direct link to the diseases caused by faulty prion proteins.
However, they insisted that the discovery might help account for some of the symptoms experienced by patients, which might be due to the failure of the proteins to do their normal job properly, rather than the damage caused by accumulation of defective prions.
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