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New Target For Retinopathy Treatment In Premature Babies Identified
Retinopathy, damage to the thin layer of cells at the back of the eyeball where light is converted into neural signals sent to the brain, is often caused by the growth of abnormal blood vessels and can lead to loss of vision. Retinopathy of prematurity (ROP) affects infants exposed to high oxygen concentrations as a part of the essential treatment they receive to allow their lungs and other organs to develop after premature birth.
"Paradoxically, it isn't high oxygen levels that damage the retina," explained first author Monica Guma, MD, PhD, of the departments of pharmacology and pathology at the UC San Diego School of Medicine. "Rather, the premature retina is first exposed to a high-oxygen environment and becomes accustomed to it. When the infant is more mature and is returned to a 'normal' oxygen environment, the retina reacts to this hypoxia – or decrease in oxygen availability – as a stress."
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