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Naturally Occurring Protein May Stop Progress of Alzheimer's Disease: Research
The study by researchers led by University of California, San Diego, showed that Memory loss, cognitive impairment, brain cell degeneration, and cell death were prevented or reversed in several animal models after treatment with BDNF.
"The effects of BDNF were potent. When we administered BDNF to memory circuits in the brain, we directly stimulated their activity and prevented cell death from the underlying disease," Nature magazine quoted Dr. Mark Tuszynski, professor of neurosciences at the UC San Diego School of Medicine and neurologist at the Veterans Affairs San Diego Health System, as saying.
Normally BDNF is produced throughout life in the entorhinal cortex, a portion of the brain that supports memory, and its production decreases in the presence of Alzheimer's disease.
For the study, BDNF gene or protein was injected in a series of cell culture and animal models, including transgenic mouse models of Alzheimer's disease; aged rats; rats with induced damage to the entorhinal cortex; aged rhesus monkeys, and monkeys with entorhinal cortex damage.
It was found that in all the above models, the treated animals demonstrated significant improvement in the performance of a variety of learning and memory tests, as compared to control groups not treated with BDNF.
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