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Fibrinogen may Inhibit Spinal Cord Regeneration
This binding, in turn, induces the activation of another receptor on the neuronal cells, called the epidermal growth factor receptor. When the second receptor is activated, it inhibits the axonal growth. Other inhibitors have been identified that use the same epidermal growth factor receptor, but this is the first blood-derived inhibitor that has been found.
The discovery may open the door to a possible strategy to improving recovery after spinal cord injury by discovering a way to block activation of neuronal receptors by fibrinogen.
Identifying the specific inhibitors that impede the repair process could provide ways to regenerate and connect the damaged nerves and initiate recovery from paralysis after spinal cord injury.
Inhibiting the damaging effects of fibrinogen on neurons may potentially facilitate repair in the nervous system after injury said Akassoglou.
A similar mechanism could be at work in other neurological diseases that result in paralysis, such as multiple sclerosis or hemorrhagic stroke, where blood vessels break and bleed into the brain. She added that such a therapeutic approach wouldnt interfere with fibrinogens essential role in coagulation, because its blood-clotting mechanism depends on binding with a different receptor.
Source-Newswise
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