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Diabetes can Be Combated by Killing "angry" Immune Cells in Fat
In the new study, the researchers tested the idea that killing those cells would reverse the inflammatory symptoms that come with obesity using a mouse model developed earlier in which the CD11c-expressing macrophages were artificially made susceptible to diphtheria toxin.
They found that treatment with the toxin not only reversed the animals' resistance to insulin, but also led to a marked decline in inflammatory signs through the body. The treated animals showed a decline in the CD11c macrophages in both fat and muscle.
" It shows that high triglycerides in muscle don't necessarily have to lead to insulin resistance as it has been thought-as long as the high lipid levels aren't accompanied by inflammation," Neels said.
The obese mice also had less fat in their livers, an important find given the epidemic of obesity-associated fatty liver disease.
Neels said that if a unique marker can be identified on the macrophages found in human fat tissue, a drug could be designed to take advantage of those features to bind and kill them,
The study is published in the October Cell Metabolism, a publication of Cell Press.
Source-ANI
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